Publication | Open Access

Ischemic-reperfusion injury in the kidney: Overexpression of colonic H+-K+-ATPase and suppression of NHE-3

InflammationRenal FunctionColonic H+-k+-atpaseReperfusion InjuryPhysiologyRenal InflammationIschemic-reperfusion InjuryAcute Kidney InjuryMedicinePotassium HomeostasisNephrologyKidney ResearchOxidative Stress