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Propranolol restores cognitive deficits and improves amyloid and Tau pathologies in a senescence-accelerated mouse model

Neurodegenerative DiseasesAlzheimer's DiseaseAgingMedicineDementiaTau PathologiesSenescence-accelerated Mouse ModelDegenerative PathologyNeurochemical BiomarkersCognitive DeficitsNeurologyNeuroscienceAging-associated DiseasePharmacology