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Specific interactions between Epac1, β-arrestin2 and PDE4D5 regulate β-adrenergic receptor subtype differential effects on cardiac hypertrophic signaling

Molecular PharmacologyCardiomyopathyCardiac MuscleMolecular PhysiologySignal TransductionSpecific InteractionsMedicinePhysiologyCardiovascular PharmacologyReceptor (Biochemistry)PharmacologyCell SignalingAlpha-adrenergic Pharmacology