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Early administration of propofol protects against endotoxin-induced acute lung injury in rats by inhibiting the TGF-β1-Smad2 dependent pathway

Acute Lung InjuryInflammatory Lung DiseaseLung InflammationImmunologyOxidative StressInflammationMolecular PharmacologyRespiratory ToxicologyEarly AdministrationPulmonary PharmacologyTissue InjuryPulmonary FibrosisVascular BiologyPharmacologyPulmonary Vascular DiseaseTgf-β1-smad2 Dependent PathwayPropofol ProtectsEndothelial DysfunctionMedicine