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Host–pathogen interactions in the development of metabolic disturbances and atherosclerosis in HIV infection: The role of CCL2 genetic variants

InflammationMetabolic SyndromeDisease MechanismMedicineHuman RetrovirusPathogenesisImmunologyHiv InfectionVascular BiologyImmune FunctionChronic Viral InfectionHivMetabolic SignalingCcl2 Genetic VariantsCardiovascular Disease PathogenesisAtherosclerosisHost–pathogen InteractionsEpidemiology