Publication | Open Access
K-Ras mutation-mediated IGF-1-induced feedback ERK activation contributes to the rapalog resistance in pancreatic ductal adenocarcinomas
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Citations
42
References
2012
Year
Tumor BiologySignaling PathwayPancreatic CancerReceptor Tyrosine KinasePancreatic Ductal AdenocarcinomasRapalog ResistanceSystems BiologyMedicineCell BiologyCell SignalingCancer Research
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