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Cardioprotection induced by olprinone, a phosphodiesterase III inhibitor, involves phosphatidylinositol-3-OH kinase-Akt and a mitochondrial permeability transition pore during early reperfusion

Signal TransductionOxysterolBiochemistryMitochondrial FunctionMedicinePhysiologyEarly ReperfusionPhosphatidylinositol-3-oh Kinase-aktPhosphodiesterase Iii InhibitorReperfusion InjuryPharmacologyOxidative Stress