Publication | Closed Access

Damage to rat liver mitochondria promoted by δ-aminolevulinic acid-generated reactive oxygen species: connections with acute intermittent porphyria and lead-poisoning

Mitochondrial FunctionBiochemistryLiver PhysiologyPhysiologyToxicologyHepatotoxicityReactive Oxygen SpecieToxicological MechanismMetabolismMedicineRedox BiologyAcute Intermittent PorphyriaPorphyriasOxidative StressHealth Sciences