Publication | Open Access

Presence or absence of a known diabetic ketoacidosis precipitant defines distinct syndromes of “A-β+” ketosis-prone diabetes based on long-term β-cell function, human leukocyte antigen class II alleles, and sex predilection

Metabolic SyndromeDiabetes ManagementDiabetes EpidemiologyAutoimmune DiseaseDisease MechanismSex PredilectionInherited Metabolic DiseaseDiabetesGenetic EpidemiologyPathologyImmunologyAutoimmunityDiabetes ComplicationsHyperglycemiaLong-term β-Cell FunctionDiabetes MellitusMedicine