Publication | Open Access

Targeting of ADAMTS5's ancillary domain with the recombinant mAb CRB0017 ameliorates disease progression in a spontaneous murine model of osteoarthritis

InflammationRheumatologySclerostinAutoimmune DiseaseSpontaneous Murine ModelImmunologyOsteoarthritisRecombinant Mab Crb0017AutoimmunityInflammatory Rheumatic DiseaseMedicineCell BiologyMusculoskeletal ResearchRheumatoid ArthritisAncillary Domain