Publication | Open Access

Three epilepsy-associated GABRG2 missense mutations at the γ+/β− interface disrupt GABAA receptor assembly and trafficking by similar mechanisms but to different extents

Synaptic PlasticityMolecular NeuroscienceSignal TransductionSimilar MechanismsReceptor (Biochemistry)Molecular BiologyDifferent ExtentsNeuroscienceMolecular NeurobiologyMedicineSynaptic SignalingCell SignalingSocial SciencesNeurogenetics