Publication | Closed Access

Rapid and profound potentiation of Apo2L/TRAIL-mediated cytotoxicity and apoptosis in thoracic cancer cells by the histone deacetylase inhibitor Trichostatin A: the essential role of the mitochondria-mediated caspase activation cascade

OncologyProfound PotentiationMedicineApoptosisCell DeathApo2l/trail-mediated CytotoxicityAnti-cancer AgentTumor SuppressorThoracic Cancer CellsPharmacologyCell BiologyTumor MicroenvironmentCancer ResearchOxidative Stress