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Dual Modulation of ERK1/2 and p38 MAP Kinase Activities Induced by Minocycline Reverses the Neurotoxic Effects of the Prion Protein Fragment 90–231

Molecular PharmacologySignal TransductionSynaptic SignalingMedicineSynaptic TransmissionNeurotoxic EffectsNeuropharmacologyPrion DiseaseDual ModulationNeuroscienceMolecular NeurobiologyNeurochemistryPharmacologyCell BiologyCell SignalingProtein PhosphorylationMinocycline Reverses