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Overexpression of cellular prion protein (PrPC) prevents cognitive dysfunction and apoptotic neuronal cell death induced by amyloid-β (Aβ1–40) administration in mice

Neurodegenerative DiseasesCognitive DysfunctionCell DeathCellular Prion ProteinDegenerative PathologyPrion DiseaseProtein MisfoldingNeuroprotectionNeuroscienceNeurologyNeurodegenerationMedicineCell Biology