Publication | Open Access

Heterozygous disruption of activin receptor–like kinase 1 is associated with increased renal fibrosis in a mouse model of obstructive nephropathy

Glomerular DiseaseRenal PathologyRenal InflammationInflammationGlomerulonephritisRenal FunctionHeterozygous DisruptionChronic Kidney DiseaseCell SignalingMouse ModelFibrosisMolecular PhysiologyRenal PathophysiologyCell BiologyIncreased Renal FibrosisUrologyRenal DiseaseGlomerulopathyMedicineNephrologyKidney Research