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Myocardial fibrosis and diastolic dysfunction in deoxycorticosterone acetate-salt hypertensive rats is ameliorated by the peroxisome proliferator-activated receptor-alpha activator fenofibrate, partly by suppressing inflammatory responses associated with the nuclear factor-kappa-b pathway

InflammationInflammatory ResponsesHypertensionHeart FailureCardiomyopathyCardiovascular DiseaseMedicinePhysiologyMyocardial FibrosisVascular BiologyNuclear Factor-kappa-b PathwayCardiovascular FunctionPharmacologyCardiologyOxidative Stress