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Decrease of current responses at human recombinant P2X3 receptors after substitution by Asp of Ser/Thr residues in protein kinase C phosphorylation sites of their ecto-domains

Molecular PhysiologySignal TransductionSer/thr ResiduesG Protein-coupled ReceptorCell SignalingMedicineReceptor Tyrosine KinaseSignaling PathwayReceptor (Biochemistry)Current ResponsesPharmacologyCell BiologyCellular PhysiologyProtein Phosphorylation