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Deletion of podocyte STAT3 mitigates the entire spectrum of HIV-1-associated nephropathy

50

Citations

24

References

2013

Year

Abstract

Development of glomerular as well as tubulointerstitial injuries in the Tg26 HIVAN model is dependent on podocyte STAT3 expression. Inhibition of STAT3 could be a potential adjunctive therapy for the treatment of HIVAN.

References

YearCitations

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