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Infarct-Sparing Effect of A <sub>2A</sub> -Adenosine Receptor Activation Is Due Primarily to Its Action on Lymphocytes

151

Citations

33

References

2005

Year

Abstract

The results indicate that A2AAR activation on bone marrow-derived cells, specifically T or B lymphocytes, is responsible for the infarct-sparing and antiinflammatory effects of ATL146e administered at the time of reperfusion after coronary occlusion.

References

YearCitations

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