Publication | Open Access

Nitric oxide attenuates cardiomyocytic apoptosis via diminished mitochondrial complex I up-regulation from cardiac ischemia-reperfusion injury under cardiopulmonary bypass

CardiomyopathyMitochondrial FunctionCardiovascular DiseaseNitric OxideReactive Nitrogen SpeciePhysiologyDiminished Mitochondrial ComplexCell DeathCardiac Ischemia-reperfusion InjuryRedox BiologyReperfusion InjuryMedicineCardiologyNitrosative StressOxidative Stress