Occurrence of leukoplakia and some other oral white lesions among 20 333 adult Swedish people

Tony Axéll

Community Dentistry And Oral Epidemiology · 1987 · 153 citations · 16 references

Concepts

TL;DR

The study calculated prevalences of oral white lesions in 20,333 adults aged 15 and older using a partly new classification. The prevalences were 5.1 % for cheek/lip biting, 1.1 % for smoker’s palate, 5.5 % for frictional lesions, 8.0 % for snuff dipper’s lesions, 6.4 % for preleukoplakia, and 3.6 % for leukoplakia, with a pooled prevalence of 24.8 % (20.1 % excluding cheek/lip biting and smoker’s palate, 13.8 % excluding weak preleukoplakic lesions); idiopathic leukoplakia was 0.7 % and tobacco‑associated 2.9 %, while homogeneous leukoplakia was 3.5 % and non‑homogeneous 0.3 %, predominantly located in commissural and buccal areas.

Abstract

Abstract Among 20 333 people aged 15 yr or above, the prevalences of oral white lesions were calculated based on a partly new classification. The total prevalences were: cheek and lip biting 5.1%, smoker's palate 1.1%, frictional white lesion 5.5%, snuff dipper's lesion 8.0%, preleukoplakia 6.4% and leukoplakia 3.6%. If all these lesions were pooled, the prevalence was 24.8% and if only the entities cheek and lip biting and smoker's palate were excluded it became 20.1%. If weak “preleukoplakic” lesions were excluded from the latter figure (he prevalence for marked whitish lesions was 13.8%. Etiologic and clinical subgroups of leukoplakia showed the following prevalences: using the etiologic subgroups, idiopathic leukoplakia 0.7% and tobacco‐associated leukoplakia 2.9%; using the clinical subgroups, homogeneous leukoplakia 3.5% and non‐homogeneous leukoplakia 0.3%. The intraoral location pattern of leukoplakias was preponderant in the commissural and buccal areas. However, the idiopathic leukoplakias showed a somewhat more even distribution and thus a more similar distribution to that of oral cancer.

References

16