A PRKAG2 mutation causes biphasic changes in myocardial AMPK activity and does not protect against ischemia

AMP-activated protein kinase mediates ischemic glucose uptake and prevents postischemic cardiac dysfunction, apoptosis, and injury

Raymond R. Russell, Ji Li, David Coven et al. · Journal of Clinical Investigation · 2004 · 723 citations · Full text

Life Sciences, Cardiology, Physiology +16

Identification of a Gene Responsible for Familial Wolff–Parkinson–White Syndrome

Michael H. Gollob, Anthony Tang, Tanya N. Gollob et al. · New England Journal of Medicine · 2001 · 674 citations · Full text

Life Sciences, Genetics, Molecular Genetics +13

Characterization of AMP-activated protein kinase γ-subunit isoforms and their role in AMP binding

Peter Cheung, Ian P. Salt, Stephen Davies et al. · Biochemical Journal · 2000 · 617 citations · Full text

Life Sciences, Biochemistry, Molecular Biology +13

Mutations in the gamma2 subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis

Edward Blair · Human Molecular Genetics · 2001 · 530 citations · Full text

Life Sciences, Cardiology, Genetics +14

Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy

Michael Arad, D. Woodrow Benson, Antonio R. Pérez‐Atayde et al. · Journal of Clinical Investigation · 2002 · 462 citations · Full text

Medicine, Cardiology, Genetics +13