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β-Ionone attenuates LPS-induced pro-inflammatory mediators such as NO, PGE2 and TNF-α in BV2 microglial cells via suppression of the NF-κB and MAPK pathway

InflammationCytokineAnti-inflammatoryImmune Cell DevelopmentMedicineImmunologyImmune RegulationBv2 Microglial CellsMapk PathwayBrain-immune InteractionImmune FunctionPharmacologyCell SignalingNeuroinflammation