Publication | Open Access

Insulin and IGF-1 improve mitochondrial function in a PI-3K/Akt-dependent manner and reduce mitochondrial generation of reactive oxygen species in Huntington’s disease knock-in striatal cells

Neurodegenerative DiseasesDevelopmental BiologyMitochondrial FunctionMitochondrial DynamicMedicineMitochondrial GenerationDegenerative PathologyMitochondrial BiologyMetabolismNeurodegenerationMetabolic SignalingReactive Oxygen SpeciesCell BiologyRedox BiologyOxidative Stress