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The role of phosphoinositide-3-kinase/Akt pathway in propofol-induced postconditioning against focal cerebral ischemia-reperfusion injury in rats

Cerebral Vascular RegulationIschemic StrokeStrokeMedicinePhosphoinositide-3-kinase/akt PathwayNeuropharmacologyPropofol-induced PostconditioningVascular BiologyBrain InjuryNeuroprotectionNeurologyAnesthesiologyCerebral Blood FlowReperfusion InjuryPharmacologyAtherosclerosisIschemic SyndromeOxidative Stress