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Involvement of soluble guanylate cyclase and calcium-activated potassium channels in the long-lasting hyporesponsiveness to phenylephrine induced by nitric oxide in rat aorta

Molecular PhysiologyNitric OxideMedicinePhysiologyEndothelial DysfunctionRat AortaVascular BiologyElectrophysiologyCardiovascular PhysiologySoluble Guanylate CyclasePharmacologyCardiologyNitrosative StressOxidative Stress