Regression of Glomerular Injury by Kallikrein Infusion in Dahl Salt-Sensitive Rats Is a Bradykinin B<sub>2</sub>-Receptor-Mediated Event

Abstract

Long-term infusion of subdepressor dose of rat urinary kallikrein attenuates functionally and morphologically the progression of renal injury in Dahl rats susceptible to salt-induced hypertension, and that the protection is mediated by stimulation of bradykinin B2 receptor.