Novel cooperation between <scp>CX</scp>3<scp>CL</scp>1 and <scp>CCL</scp>26 inducing <scp>NK</scp> cell chemotaxis via <scp>CX</scp>3<scp>CR</scp>1: a possible mechanism for <scp>NK</scp> cell infiltration of the allergic nasal tissue

Abstract

Our results highlight a novel role for CX3CR1 in NK cell migration that may contribute to the NK cell trafficking to the allergic upper airway. This could be mediated largely by CX3CL1 and CCL26 stimulation of the tyrosine kinase pathway.