Publication | Open Access

Tumor necrosis factor-α produced in cardiomyocytes mediates a predominant myocardial inflammatory response to stretch in early volume overload

InflammationCardiac MuscleCardiomyopathyHeart FailureCardiovascular DiseaseMyocardial InfarctionPhysiologyImmunologyCardiomyocytes MediatesPathologyEarly Volume OverloadCardiovascular FunctionMedicineCardiologyTumor Necrosis Factor-αCardiac Pathology