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ALS-Linked Cu/Zn–SOD Mutation Increases Vulnerability of Motor Neurons to Excitotoxicity by a Mechanism Involving Increased Oxidative Stress and Perturbed Calcium Homeostasis

Neurodegenerative DiseasesAmyotrophic Lateral SclerosisMotor Control DisordersNeurophysiologyMotor NeuronsPhysiologySocial SciencesNeuroscienceNeurologyNeurodegenerationMolecular NeurobiologyMedicineSynaptic SignalingPerturbed Calcium HomeostasisOxidative Stress