Publication | Open Access

Chronic Akt activation attenuated lipopolysaccharide-induced cardiac dysfunction via Akt/GSK3β-dependent inhibition of apoptosis and ER stress

InflammationHeart FailureMolecular PhysiologyCardiovascular DiseaseChronic Akt ActivationCell SignalingMedicinePhysiologyApoptosisCardiovascular PharmacologyCell DeathDisease PathophysiologyEr StressCardiovascular ToxicityPharmacologyCardiologyLipopolysaccharide-induced Cardiac DysfunctionOxidative Stress