Publication | Closed Access

New insights into the molecular mechanisms underlying sensitivity/resistance to the atypical retinoid ST1926 in acute myeloid leukaemia cells: The role of histone H2A.Z, cAMP-dependent protein kinase A and the proteasome

Histone ModificationsChromatinHematological MalignancyChromatin RemodelingMixed-phenotype Acute LeukemiaAtypical Retinoid St1926Molecular BiologyEpigeneticsNew InsightsHistone H2a.zMedicineCell BiologyCell SignalingTumor BiologyMyeloid Neoplasia