Homoisoflavanone inhibits retinal neovascularization through cell cycle arrest with decrease of cdc2 expression

Vascular Endothelial Growth Factor in Ocular Fluid of Patients with Diabetic Retinopathy and Other Retinal Disorders

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Oxygen-induced retinopathy in the mouse.

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Mechanisms of G2 Arrest in Response to Overexpression of p53

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Thrombospondin‐1–deficient mice exhibit increased vascular density during retinal vascular development and are less sensitive to hyperoxia‐mediated vessel obliteration

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Anti-Angiogenic Activity of a Homoisoflavanone from<i>cremastra appendiculata</i>

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