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Catalpol suppresses advanced glycation end-products-induced inflammatory responses through inhibition of reactive oxygen species in human monocytic THP-1 cells

InflammationCytokineAnti-inflammatorySignal TransductionReactive Oxygen SpeciesLung InflammationMedicineImmunologyEndothelial DysfunctionCell DeathChronic InflammationPharmacologyCell BiologyCatalpol SuppressesOxidative Stress