Antibodies to Glutamic Acid Decarboxylase Reveal Latent Autoimmune Diabetes Mellitus in Adults With a Non—Insulin-Dependent Onset of Disease

Leif Groop, Paul Zimmet, Merrill J. Rowley, W J Knowles

Diabetes · 1993 · 562 citations · 11 references

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TL;DR

Adults with diabetes who initially present as non‑insulin‑dependent can later become insulin‑dependent, suggesting that some cases involve autoimmune β‑cell destruction similar to type 1 diabetes. The authors examined 102 adults (>35 yr) with ≥6 mo of non‑ketotic, non‑insulin‑dependent diabetes, classified them by C‑peptide into insulin‑deficient and non‑deficient groups, and measured antibodies to GAD, islet cell cytoplasm, thyroid, and gastric antigens. Anti‑GAD antibodies were present in 76 % of the insulin‑deficient group versus 12 % of the non‑deficient group, a markedly higher prevalence than for ICAs, indicating that anti‑GAD testing can uncover latent autoimmune diabetes in adults initially presenting as NIDDM and has implications for early intervention and accurate classification.

Abstract

The classification of adults with diabetes mellitus can be invalidated by patients who initially present as NIDDM but who later become frankly insulin dependent. In some of these, the pathogenesis could be similar to that in IDDM, namely autoimmune destruction of the pancreatic β-cells. We studied 102 patients >35 yr of age at diabetes onset who had initially been nonketotic and non-insulin-dependent for ≥6 mo. They were classified according to glucagonstimulated C-peptide levels into an insulin-deficient group (n = 33) and a non-insulin-deficient group (n = 69). We measured antibodies to GAD, islet cell cytoplasm, thyroid antigens, and gastric parietal cells in both groups. Anti-GAD was significantly higher in the insulin deficient group, 76% (25 of 33), than in the non-insulin deficient group, 12% (8 of 69), and this difference was substantially greater than that shown for ICAs. Thus, in a proportion of adults who present with NIDDM, a slowly evolving autoimmune insulitis can be revealed by testing for anti-GAD. This could have important connotations not only for early intervention, but also for the correct classification of diabetes.

References

11