Publication | Open Access

N-Methyl-d-aspartate Receptor- and Calpain-mediated Proteolytic Cleavage of K+-Cl− Cotransporter-2 Impairs Spinal Chloride Homeostasis in Neuropathic Pain

Molecular PharmacologyMolecular PhysiologyHealth SciencesBiochemistryPain MedicineMedicinePhysiologyReceptor (Biochemistry)Neuropathic PainIon ChannelsNeuropharmacologyMolecular PainPain ManagementCalpain-mediated Proteolytic CleavagePain MechanismPharmacologyN-methyl-d-aspartate Receptor-Pain Research