Amelioration of Ethanol‐Induced Neurotoxicity in the Neonatal Rat Central Nervous System by Antioxidant Therapy

Abstract

These results suggest that ethanol-related cerebellar damage during this early postnatal period may be related to oxidative stress processes or the insufficiency of protective antioxidants. Thus, antioxidant treatment may represent a possible therapy for preventing or ameliorating the central nervous system (CNS) damage seen in the fetal alcohol syndrome.