C-Reactive Protein Co-Expresses with Tumor Necrosis Factor-α in the Myocardium in Human Dilated Cardiomyopathy

Abstract

Cardiac expression of CRP with TNF-alpha may function as a proinflammatory mediator in DCM and may be related to the clinical severity of DCM. Expression of both of these proteins was decreased in DCM patients receiving spironolactone and either ACEIs or ARBs.