Publication | Closed Access

Spontaneous epileptiform activity mediated by GABAA receptors and gap junctions in the rat hippocampal slice following long-term exposure to GABAB antagonists

Synaptic PlasticityGap JunctionsNeurophysiologyNeuroanatomyMedicineNeurotransmitterNeuropeptide ReceptorNeuropharmacologySocial SciencesNeurologyNeurotransmissionNeuroscienceCentral Nervous SystemPharmacologyNeurochemistryGabaa ReceptorsGabab Antagonists