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Interleukin‐28 gene polymorphisms may contribute to <scp>HB</scp>sAg persistence and the development of <scp>HB</scp>eAg‐negative chronic hepatitis B

13

Citations

38

References

2014

Year

Abstract

These results suggest that IL-28B polymorphisms may contribute to HBsAg persistence and the development of the inactive HBsAg carrier state.

References

YearCitations

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