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Blood-brain barrier breakdown and increased intercellular adhesion molecule (ICAM-1/CD54) expression after Semliki Forest ( A7) virus infection facilitates the development of experimental allergic encephalomyelitis

Autoimmune DiseaseAllergyVirus InfectionSemliki ForestMedicineImmunologyViral PathogenesisAntiviral ResponseImmunologic MechanismAutoimmunityBrain-immune InteractionImmunopathologyNeuroimmunologyBlood-brain Barrier Breakdown