Publication | Open Access
Innate <scp>I</scp>mmunity to <scp><i>C</i></scp><i>ampylobacter jejuni</i> in <scp>G</scp>uillain‐<scp>B</scp>arré <scp>S</scp>yndrome
41
Citations
46
References
2015
Year
These results suggest that a strong response to TLR4 stimulation is a critical host condition for the development of GBS after an infection with C. jejuni.
| Year | Citations | |
|---|---|---|
Page 1
Page 1