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Innate <scp>I</scp>mmunity to <scp><i>C</i></scp><i>ampylobacter jejuni</i> in <scp>G</scp>uillain‐<scp>B</scp>arré <scp>S</scp>yndrome

41

Citations

46

References

2015

Year

Abstract

These results suggest that a strong response to TLR4 stimulation is a critical host condition for the development of GBS after an infection with C. jejuni.

References

YearCitations

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