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Indomethacin and epinephrine effects on outflow facility and cyclic adenosine monophosphate formation in monkeys.

19

Citations

67

References

1996

Year

Abstract

Approximately half of the epinephrine-induced facility increase is inhibited by indomethacin, but it is unclear whether the indomethacin-inhibitable fraction is mediated by epinephrine-stimulated prostanoid production or release.

References

YearCitations

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