Publication | Closed Access
Upregulation of K <sub>2P</sub> 3.1 K <sup>+</sup> Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation
191
Citations
37
References
2015
Year
Enhancement of atrium-selective K(2P)3.1 currents contributes to APD shortening in patients with chronic AF, and K(2P)3.1 channel inhibition reverses AF-related APD shortening. These results highlight the potential of K(2P)3.1 as a novel drug target for mechanism-based AF therapy.
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