Complementary action of granulocyte macrophage colony-stimulating factor and interleukin-17A induces interleukin-23, receptor activator of nuclear factor-κB ligand, and matrix metalloproteinases and drives bone and cartilage pathology in experimental arthritis: rationale for combination therapy in rheumatoid arthritis

Publication | Open Access

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References

2015

Year

Annemarie van Nieuwenhuijze, Fons AJ van de Loo, Birgitte Walgreen, Miranda B. Bennink, Monique M. Helsen, Liduine van den Bersselaar, Ian P. Wicks, Wim B. van den Berg, Marije I. Koenders

Arthritis Research & Therapy

ImmunologyImmune RegulationPathologyComplementary ActionImmune SystemInflammatory ArthritisInflammationRheumatoid DisorderCartilage DegenerationInflammatory Rheumatic DiseaseRheumatoid ArthritisRheumatologyAutoimmune DiseaseRheumatic DiseasesChronic InflammationInflammatory DiseaseCytokineCombination TherapyMedicineSystemic Juvenile Idiopathic Arthritis