Role of Oxidative Tissue Injury in the Pathophysiology of Experimentally Induced Equine Laminitis: A Comparison of 2 Models

Abstract

These results do not support a prominent role for oxidative stress at examined time points in CHO-overload laminitis and support transient oxidative stress in the BWE model. Tissue oxidation does not appear to be a central early pathophysiologic event in CHO-associated laminitis.