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Mouse models for Friedreich ataxia exhibit cardiomyopathy, sensory nerve defect and Fe-S enzyme deficiency followed by intramitochondrial iron deposits

Intramitochondrial Iron DepositsSensory Nerve DefectIron MetabolismPhysiologyMouse ModelsDegenerative DiseaseDegenerative PathologyNeurologyNeuroscienceNeuropathologyMedicineOxidative Stress