Thomas F.E. Barth - Concepedia

Author

Thomas F.E. Barth

Also Known As

Barth T, Barth, T F, Barth, Thomas, Barth, Thomas F. E., T Barth, T F Barth, T F E Barth, T. Barth, T. F. Barth, T. F. E. Barth

452

Publications

12.9K

Citations

H-Index

163

Concepts

All Affiliations

Universität Ulm University Hospital Ulm Heidelberg University German Cancer Research Center University Hospital Schleswig-Holstein

About

Thomas F.E. Barth is an author at Universität Ulm specializing in medicine, pathology, and cell biology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	29	53	2.7K
2	Health Sciences	7	7	423
3	Engineering	2	2	139
4	Natural Sciences	1	1	25
5	Social Sciences	1	1	75

Thomas F.E. Barth

159

Publications

11.9K

Citations

H-Index

Page 1

	Year	Citations
A Biologic Definition of Burkitt's Lymphoma from Transcriptional and Genomic Profiling Michael Hummel, Stefan Bentink, Hilmar Berger, New England Journal of Medicine	2006	1K
T-lymphocyte Infiltration in Visceral Adipose Tissue Ulrich Kintscher, Martin Hartge, Anna Foryst‐Ludwig, Arteriosclerosis Thrombosis and Vascular Biology	2008	672
SARS-CoV-2 infects and replicates in cells of the human endocrine and exocrine pancreas Janis A. Müller, Rüdiger Groß, Carina Conzelmann, Nature Metabolism	2021	552
MYC status in concert with BCL2 and BCL6 expression predicts outcome in diffuse large B-cell lymphoma Heike Horn, Marita Ziepert, Claudia Becher, Blood	2013	540
MYC stimulates EZH2 expression by repression of its negative regulator miR-26a Sandrine Sander, Lars Bullinger, Kay Klapproth, Blood	2008	395
Prognostic Value of Ki-67 Index, Cytology, and Growth Pattern in Mantle-Cell Lymphoma: Results From Randomized Trials of the European Mantle Cell Lymphoma Network Eva Hoster, Andreas Rosenwald, Françoise Berger, Journal of Clinical Oncology	2016	344
Interleukin 21–Induced Granzyme B–Expressing B Cells Infiltrate Tumors and Regulate T Cells Stefanie Lindner, Karen Dahlke, Kai Sontheimer, Cancer Research	2013	336
Mutations of the tumor suppressor gene SOCS-1 in classical Hodgkin lymphoma are frequent and associated with nuclear phospho-STAT5 accumulation Marc A. Weniger, Ingo Melzner, Christiane K. Menz, Oncogene Lymphoid NeoplasiaPathologyClassical Hodgkin LymphomaTumor SuppressorCancer Genetics	2006	316
Reticular dysgenesis (aleukocytosis) is caused by mutations in the gene encoding mitochondrial adenylate kinase 2 Ulrich Pannicke, Manfred Hönig, Isabell Hess, Nature Genetics Developmental BiologyMitochondrial BiogenesisMitochondrial FunctionCell DivisionReticular Dysgenesis	2008	239
Classical Hodgkin lymphoma is characterized by recurrent copy number gains of the short arm of chromosome 2 Stefan Joos, Christiane K. Menz, Gunnar Wrobel, Blood	2002	234

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