Maha Abdellatif - Concepedia

Author

Maha Abdellatif

Rutgers, The State University of New Jersey ORCID

Also Known As

D Olver, M Abdellatif, M M Abdellatif, M. Abdellatif, M. M. Abdel‐Latif, Maha Abdellatif, Maha M. Abdellatif

112

Publications

9.6K

Citations

H-Index

Concepts

All Affiliations

Rutgers, The State University of New Jersey Rutgers New Jersey Medical School Baylor College of Medicine University Hospital, Newark New Jersey Institute of Technology

About

Maha Abdellatif is an author at Rutgers, The State University of New Jersey specializing in medicine, cell biology, and physiology.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	23	28	4.3K
2	Natural Sciences	6	6	362
3	Engineering	2	2	327
4	Health Sciences	2	2	446
5	Public Health	1	1	139

Maha Abdellatif

Publications

Citations

H-Index

Page 1

	Year	Citations
Distinct Roles of Autophagy in the Heart During Ischemia and Reperfusion Yutaka Matsui, Hiromitsu Takagi, Xueping Qu, Circulation Research MitophagyHeart FailureCardiovascular FunctionCellular PhysiologyOxidative Stress	2007	1.5K
MicroRNAs Play an Essential Role in the Development of Cardiac Hypertrophy Danish Sayed, Chull Hong, Ieng-Yi Chen, Circulation Research	2007	784
Downregulation of MiR-199a Derepresses Hypoxia-Inducible Factor-1α and Sirtuin 1 and Recapitulates Hypoxia Preconditioning in Cardiac Myocytes Shweta Rane, Minzhen He, Danish Sayed, Circulation Research Cardiac MuscleCell DeathOxidative StressTranscriptional RegulationRedox Regulator	2009	573
Endogenous Drp1 Mediates Mitochondrial Autophagy and Protects the Heart Against Energy Stress Yoshiyuki Ikeda, Akihiro Shirakabe, Yasuhiro Maejima, Circulation Research	2014	560
MicroRNA-21 Targets Sprouty2 and Promotes Cellular Outgrowths Danish Sayed, Shweta Rane, Jacqueline Lypowy, Molecular Biology of the Cell	2008	353
Simvastatin Induces Regression of Cardiac Hypertrophy and Fibrosis and Improves Cardiac Function in a Transgenic Rabbit Model of Human Hypertrophic Cardiomyopathy Rajni V. Patel, Sherif F. Nagueh, Natalia Tsybouleva, Circulation	2001	338
MicroRNA-21 Is a Downstream Effector of AKT That Mediates Its Antiapoptotic Effects via Suppression of Fas Ligand Danish Sayed, Minzhen He, Chull Hong, Journal of Biological Chemistry Antiapoptotic EffectsHeart FailureEngineeringImmunologyCell Death	2010	314
<i>miR-1</i> Overexpression Enhances Ca <sup>2+</sup> Release and Promotes Cardiac Arrhythmogenesis by Targeting PP2A Regulatory Subunit B56α and Causing CaMKII-Dependent Hyperphosphorylation of RyR2 Dmitry Terentyev, Andriy E. Belevych, Radmila Terentyeva, Circulation Research	2009	279
Mitochondrial complex II is a source of the reserve respiratory capacity that is regulated by metabolic sensors and promotes cell survival Jessica Pfleger, Miao He, Maha Abdellatif Cell Death and Disease	2015	254
Activation and function of cyclin T–Cdk9 (positive transcription elongation factor-b) in cardiac muscle-cell hypertrophy Motoaki Sano, Maha Abdellatif, Hidemasa Oh, Nature Medicine Cardiac MuscleMuscle PhysiologyTranscriptional RegulationSignal TransductionMolecular Physiology	2002	237

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