Cornelia Dietrich - Concepedia

Author

Cornelia Dietrich

Johannes Gutenberg University Mainz ORCID

Also Known As

C Dietrich, C. Dietrich, Colton Dietrich, Cornelia Dietrich, Dietrich, Cornelia

Publications

1.8K

Citations

H-Index

Concepts

All Affiliations

Johannes Gutenberg University Mainz University Medical Center of the Johannes Gutenberg University Mainz Czech Academy of Sciences, Institute of Biophysics Veterinary Research Institute Karlsruhe Institute of Technology

About

Cornelia Dietrich is an author at Johannes Gutenberg University Mainz specializing in medicine, cell biology, and cell signaling.

Top Concepts

#	Concept	H-Index	Publications	Citations
1	Medicine	18	22	1.1K
2	Cell Biology	15	18	931
3	Biochemistry	4	4	174
4	Natural Sciences	2	2	99
5	Health Sciences	1	1	75

Cornelia Dietrich

Publications

1.3K

Citations

H-Index

Page 1

	Year	Citations
Aryl hydrocarbon receptor activation by cAMP vs. dioxin: Divergent signaling pathways Barbara Oesch‐Bartlomowicz, Andrea Huelster, Oliver Wiss, Proceedings of the National Academy of Sciences	2005	178
TCDD induces c-jun expression via a novel Ah (dioxin) receptor-mediated p38–MAPK-dependent pathway Dagmar Faust, Heike Dürk, Siva K. Kolluri, Oncogene InflammationMolecular PharmacologyC-jun ExpressionSignal TransductionSignaling Pathway	2005	91
The aryl hydrocarbon receptor-dependent deregulation of cell cycle control induced by polycyclic aromatic hydrocarbons in rat liver epithelial cells Zdeněk Andrysík, Jan Vondráček, Miroslav Machala, Mutation research. Fundamental and molecular mechanisms of mutagenesis BiochemistryMedicineLiver PhysiologyCell Cycle ControlToxicology	2006	81
TCDD deregulates contact inhibition in rat liver oval cells via Ah receptor, JunD and cyclin A Dagmar Faust, Ilona Schreck, Albert L. Ruff, Oncogene InflammationSignal TransductionSignaling PathwayMedicineLiver Physiology	2007	77
p38α MAPK is required for contact inhibition Dagmar Faust, Ignacio Dolado, Ana Cuadrado, Oncogene Molecular PharmacologyMolecular PhysiologySignal TransductionSignaling PathwayMedicine	2005	75
p53-dependent cell cycle arrest induced by N-acetyl-L-leucinyl-L-leucinyl-L-norleucinal in platelet-derived growth factor-stimulated human fibroblasts. Cornelia Dietrich, Thomas Bartsch, F Schanz, Proceedings of the National Academy of Sciences Cell DeathCell ProliferationCell CycleTumor BiologyCell Regulation	1996	75
t-BuOOH induces ferroptosis in human and murine cell lines Christine Wenz, Dagmar Faust, Berenike Linz, Archives of Toxicology Signal TransductionDisease MechanismCell SignalingMedicineImmunology	2017	71
The histone deacetylases HDAC1 and HDAC2 are required for the growth and survival of renal carcinoma cells Nicole Kiweler, Boris Brill, Matthias Wirth, Archives of Toxicology Histone ModificationsChromatinUrologyGenitourinary CancerChromatin Remodeling	2018	71
Differences in the mechanisms of growth control in contact-inhibited and serum-deprived human fibroblasts Cornelia Dietrich, Katja Wallenfang, Franz Oesch, Oncogene MechanobiologyFibrosisDevelopmental BiologyGrowth ControlSerum-deprived Human Fibroblasts	1997	68
p16INK4 mediates contact-inhibition of growth Raimund Wieser, Dagmar Faust, Cornelia Dietrich, Oncogene Signal TransductionSignaling PathwayNatural SciencesMolecular BiologyCytoskeleton	1999	67

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